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[1] Wu B, Fu Z, Wang X, et al.A narrative review of diabetic bone disease: Characteristics, pathogenesis, and treatment[J]. Front Endocrinol (Lausanne), 2022, 13: 1052592. [2] Bai J, Gao Q, Wang C, et al.Diabetes mellitus and risk of low-energy fracture: a meta-analysis[J]. Aging Clin Exp Res, 2020, 32(11): 2173-2186. [3] Si Y, Wang C, Guo Y, et al.Prevalence of osteoporosis in patients with type 2 diabetes mellitus in the Chinese mainland: a systematic review and meta-analysis[J]. Iran J Public Health, 2019, 48(7): 1203-1214. [4] 蔡俊民,周后德. 免疫性骨质疏松及其研究进展[J].中华内分泌代谢杂志,2022,38(7):558-566. [5] Read K, Powell M, Sreekumar B, et al.In vitro differentiation of effector CD4+ T helper cell subsets[J]. Methods Mol Biol, 2019, 1960: 75-84. [6] Prasad T, Arjunan D, Pal R, et al.Diabetes and osteoporosis[J]. Indian J Orthop, 2023, 57(Suppl 1): 209-217. [7] Chiodini I, Catalano A, Gennari L, et al.Osteoporosis and fragility fractures in type 2 diabetes[J]. Diabetes Res, 2020, 2020: 9342696. [8] Sheu A, Greenfield JR, White CP, et al.Contributors to impaired bone health in type 2 diabetes[J]. Trends Endocrinol Metab, 2023, 34(1): 34-48. [9] Li H, Wen Y, Liu P, et al.Characteristics of bone metabolism in postmenopausal women with newly diagnosed type 2 diabetes mellitus[J]. Clin Endocrinol (Oxf), 2021, 95(3): 430-438. [10] Nishinka T, Hatipoglu OF, Wake H, et al.Different modulation of STING/TBK1/IRF3 signaling by advanced glycation end products[J]. Arch Biochem Biophys, 2023, 750: 109808. [11] 李彬,伍军伟,谭欣. 胰岛素样生长因子-1 表达水平在 2 型糖尿病并发骨质疏松症中的临床意义[J].热带医学杂志,2021,21(6):774-776,781. [12] Chen X, Famurewa A, Tang J, et al.Hyperoside attenuates neuroinflammation, cognitive impairment and oxidative stress via suppressing TNF-α/NF-κB/caspase-3 signaling in type 2 diabetes rats[J]. Nutr Neurosci, 2022, 25(8): 1774-1784. [13] 唐洁,蒲姝陶. 辅助性T 细胞亚群分化机制及临床意义研究进展[J]. 巴楚医学,2020,3(4):103-107,116. [14] Garcia-Jacobo RE, Bergamin LS, Vultaggio-Poma V, et al.The purinergic landscape of type 2 diabetes mellitus[J]. Molecules, 2022, 27(6): 1838. [15] Duddu AS, Majumdar SS, Sahoo S, et al. Emergent dynamics of a three-node regulatory network explain phenotypic switching and heterogeneity: a case study of Th1/Th2/Th17 cell differentiation[J]. Mol Biol Cell, 2022, 33(6): ar46. [16] Kar S, Gupta R, Malhotra R, et al.Interleukin-9 facilitates osteoclastogenesis in rheumatoid arthritis[J]. Int J Mol Sci, 2021, 22(19): 10397. [17] Wang J, Zhao X, Wan Y.Intricacies of TGF-β signaling in Treg and Th17 cell biology[J]. Cell Mol Immunol, 2023, 20(9): 1002-1022. [18] Cafferata E, Castro-Saavedra S, Fuentes-Barros G, et al.Boldine inhibits the alveolar bone resorption during ligature-induced periodontitis by modulating the Th17/Treg imbalance[J]. Periodontol, 2021, 92(1): 123-136. [19] Fischer L, Herkner C, Kitte R, et al.Foxp3+ regulatory T cells in bone and hematopoietic homeostasis[J]. Front Endocrinol (Lausanne), 2019,10: 578. [20] Miyazaki Y, Nakayamada S, Kubo S, et al.Th22 cells promote osteoclast differentiation via production of IL-22 in rheumatoid arthritis[J]. Front Immunol, 2018, 9: 2901. [21] Wei X, Niu X.T follicular helper cells in autoimmune diseases[J]. Autoimmun, 2023, 134: 102976.
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